Moreover, in PC3 cell xenograft tumor tissues, C10 therapy inhibited tumor development and upregulated PKCδ. These results suggest that C10 treatment induces the PKCδ/JNK pathway, therefore activating Caspase-3 and inducing the cleavage of PARP and gasdermin E to execute apoptosis and cell-lytic pyroptosis in prostate cancer cells. Some Negative effects https://oss-12816745434.blog-eye.com/31185680/5-essential-elements-for-c646